The objective of the paper to analyse the linkages of growth of health expenditure and health outcome in India from 1991-2021. After the structural adjustment programme, healthcare expenditure witnessed a deceleration worldwide with the extreme situation faced by developing countries. So, given the emerging challenges of health problems in the developing world, the financing mechanism of public health services requires more attention in catering healthcare needs of the population more adequately and effectively. This study tried to examine the nexus between health outcomes and health expenditure in India. This study is purely based on secondary sources of data from the Reserve Bank of India, the World Bank Database, and various government reports. The trend of IMR and LEB in India during the period post-reform the LEI has an increasing trend slowly over the period whereas IMR has a declining trend. The Granger-causality Wald test is conducted to establish whether health expenditure affects GDP and vice-versa. Health spending has a positive impact on India's GDP. The correlation between health spending and health outcomes like-LEB, IMR, are significant and Positive for life expectancy at birth and negative for infant mortality rate, the short-run impact on life expectancy with health expenditure is positive and significant, while there is a negative impact of IMR with health expenditure in long-run. This study highlighted that more awareness of the health of the people is necessary if sustainable growth is pursued.
Research on healthcare spending, outcomes, and GDP has provided conflicting results. A study conducted by Balaji (2011)1 identified no evidence of a long-term causal relationship between healthcare spending and economic growth. In contrast, Elmi and Sadeghi (2012)2 and Mehrara and Musai (2011)3 indicate a short-term causal nexus between healthcare spending and GDP. Amiri and Ventelou (2010)4 also emphasize that there is bidirectional causality between economic growth and healthcare spending. Hooda (2014)5 investigated how various forms of decentralized governance affected infant and child mortality rates in rural India across states and how they affected the effectiveness of spending on rural health. The findings demonstrated that states with high political and fiscal decentralization have a more significant effect on lowering infant mortality than states with high budgetary but low political decentralization levels, suggesting that fiscal decentralization's effectiveness rises with political decentralization.
Healthcare spending is crucial for both short- and long-term gains. Reeves et al. (2013)6 and Llori and Babatola (2017)7. According to Karaman et al. (2020)8, one of the key elements of long-term sustainable economic development is good health, a significant component of human capital. According to Karim (2016)9, Romer (2012)10, and Sen (2014)11, According to the neo-classical growth model, a healthier and educated labour force raises per capita income for people and their families, increasing the value of human life. Improvements in medical facilities and opportunities to develop human capital can result from expenditure on health care, which boosts productivity and economic performance. Albulescu et al. (2017)12, Raghupathi (2015)13. An important health effect of increased public spending on emergency aid, curative care, and nutrition and immunization programs is reduced mortality. There have been contradictions in the literature about the relationship between healthcare spending and better health outcomes. Health spending is associated with two health outcomes: more extended life expectancy and reduced child mortality (Karaman et al., Chiristopolos)14, Kim, and Lane)15. For example, one study demonstrated that healthcare spending in OECD countries positively impacted health outcomes, including life expectancy and maternal and neonatal mortality (Karaman). (Kim and Lane)15 studied 17 OECD countries from 1973to 2000, employing life expectancy at birth and infant mortality as health outcome indicators, and found a positive relationship between health spending and health outcomes. An additional study (Onofrei et al.)16 showed that increasing health spending increases life expectancy and decreases infant mortality. However, using Spearman's correlation technique, other studies (Lippi et al (2016)17. and Mackenbach,1991)18 demonstrated no connection between mortality rates in European nations and healthcare spending. Some researchers found no correlation between health spending and health status (Deshpande,) 19. At the same time, another study (van et al.) 20 showed that it is difficult to establish a relationship between healthcare spending and health outcomes (life expectancy). The hypothesis indicates that to improve health outcomes, health investment is essential. According to the literature, a country's health spending level is a crucial indicator of its degree of health investment. This originates from the fact that, through the administration and delivery of health care services, the government may use expenditure on healthcare, particularly public health spending, as a significant policy weapon in any economy. Consequently, empirical researchers have worked to confirm how health spending influences health outcomes.
The inquiry concerns whether and to what degree health spending affects health outcomes. The importance of health spending on health outcomes has been studied using the Grossman health capital model, was developed based on Grossman's 1972 seminar, Health Capital and the Demand for Healthcare. Health spending is one of the inputs into the "production" of healthcare under both models, along with other socioeconomic determinants of health that are equally important.
Objectives
Hypotheses
H1: There is no linkage between health Expenditure and Life Expectancy.
H2: There is no linkage between health expenditure and infant mortality in India.
Data Methodology
This study has compiled secondary sources of data from - macro indicators of RBI bulletin and World Bank database etc. Variables of the study: Healthcare expenditure in (Revenue and Capital), account, Health outcome (Life Expectancy at birth, Infant Mortality Rate.)
Study Period: 1991-2021
Statistical tools and Methods:
Stationarity Test (Unit Root Test)
A stationary time series is essential for avoiding spurious causation in time series analysis. Spurious causality is a high correlation between two non-stationary time series variables with no causal relationship. Traditional regression techniques are incorrect when the series contains a unit root because they become erroneous, and vice versa. A time series is considered stationary (i.e., without a unit root) if its mean, variance, and autocorrelation do not change over time. If not, it is known as a unit root or a non-stationary time series (Gujarati 2009).
A time series of order zero or I (0) will be integrated if the stationarity test indicates that it is stationary at level (without differencing, i.e., Yt). To avoid spurious correlation, a series of order one or I (1) will be integrated if the test indicates that it is stationary at the first difference (i.e., Yt - Yt-1) for each variable. We use a parametric approach that includes the augmented Dickey and Fuller (ADF) test, which was pioneered by Dickey and Fuller (1979), and the Phillips-Perron (PP) unit root tests.
The ADF test is based on the following regression equation:
The equation is ∆Yt = Yt - Yt-1, with α1 as the constant term, t as the time trend, ∆ the first difference operator, n as the optimal number of lags, and εt as the pure white noise term. The null Hypothesis for the ADF test is HO: δ = 0, indicating that the time series is non- stationary (with a unit root).
The Phillips-Perron (PP) test is used to assess the stationarity of each series variable in addition to the Augmented Dickey-Fuller (ADF) test. Unlike the ADF test, the PP test does not require a lagged difference term to determine the level of serial correlation. Therefore, the PP test is based on the following regression equation:
The PP test's null Hypothesis, similar to the ADF test, is HO: δ = 0, indicating that the time series is non-stationary (with a unit root).
After analyzing the unit root testing, the next step is to choose the lag length for co- integration because the number of lags captures the dynamics of the series. There are different criteria for the selection of optimal lag length. The requirements help in selecting the appropriate lag order for the model. The lag order chosen is indicated by "*" in the last three columns for each criterion. For example, in the "AIC" column, the lag order with the lowest AIC value is selected as the appropriate lag order based on the AIC criterion. In the "LogL" column, higher values suggest a better model fit. The lag order chosen has the highest LogL value. The "LR" value compares the likelihood of the current model with the previous one. The quality of the current model increases with the LR value. Lower values in the "FPE," "AIC," "SC," and "HQ" columns indicate better model performance based on these criteria.
In econometrics, it is a statistical technique used to ascertain whether variables have a long-term relationship, particularly when those variables are integrated in different orders (i.e., I(0) or I(1)) but not I(2). When working with variables with heterogeneous integration orders or small sample sizes, this test is commonly uses the boundaries test to ascertain a co-integration relationship between the variables. The researchers provide the critical values for the bound test, which can be compared to the computed F-statistic based on the number of variables and the significance level.
Regression analysis is a situation in which the variance of the error terms, also known as residuals, varies at different levels of the independent variable. But, the spread or "scatter" of residuals changes when the value of the independent variable changes. This is contrary to homoscedasticity, one of the fundamental tenets of ordinary least squares (OLS) regression, which asserts that the variance of errors is constant.
Heteroscedasticity can affect the efficiency of the regression estimates, leading to inefficient parameter estimates, biased standard errors, and unreliable hypothesis tests (e.g., t-tests, F-tests). Therefore, detecting and addressing heteroscedasticity is an important part of regression analysis. To understand heteroscedasticity, we look at the model:
Where:
Normality Test
A statistical method determines whether a given dataset has a normal distribution (bell curve). The null Hypothesis in a normality test typically states that the data is distributed normally. When the p-value is significant (typically p < 0.05), the data does not fit a normal distribution, and the null Hypothesis is rejected. A high p-value, on the other hand, indicates that the data may have a normal distribution and that the null Hypothesis cannot be dismissed.
A standard statistical method for determining if a serial correlation exists in the residuals of a linear regression model is the Breusch-Godfrey LM test. This test is frequently utilized because it may detect higher-order serial correlation in addition to first-order autocorrelation and does not need the error terms to be normally distributed.
The Breusch-Godfrey LM test is based on the Lagrange multiplier (LM) principle, a method for assessing the significance of a group of constraints in a model. In this case, the rule is that the residuals of the regression model should not be serially correlated.
Linear regression model: yt = β0 + β1xt + ϵt
Where:
Autoregressive Distributed Lag (ARDL) Approach
Asymptotic critical values are proposed in two sets by Pesaran et al. (2001). In the first set, all underlying variables are assumed to be integrated at the level, or I (0). On the other hand, in the second set, I(1), all underlying variables are expected to be integrated into order one, indicating a long-term relationship between the variables. This study tests for co-integration between the variables using the Autoregressive Distributed Lag (ARDL) method. This approach is superior to other conventional methods, including those put forth by Engle and Granger (1987), Johansen (1991), and Gregory and Hansen (1996). The result cannot be inferred if the F-statistics in between the lower and upper critical bounds. The null Hypothesis cannot be refuted. if the F-statistic is less than the lower critical bound. The model's long- and short-term coefficients can be estimated if the variables have a long-term relationship.
The first step is to determine whether the variables have a long-term relationship. The second step is to estimate the long-run coefficients of the model. The ARDL Long Run Form and Bound test determines whether the variables have a long-term relationship. The null Hypothesis that there is no long-term relationship among the coefficients of lagged variables, πi, is examined by the well-known F-statistic.
An ARDL (Autoregressive-distributed lag) is a parsimonious infinite lag-distributed model. The term “autoregressive” shows that along with getting explained by the xt, yt also gets explained by its lag. The equation of ARDL (m, n) is as follows:
Y = Health Outcome Variable (LEB, IMR)
LHEXP= Log of total health expenditure
εt is the disturbance term, and
βi’s are coefficients for short-run and
αi’s are coefficients for long-run relationships.
In time series analysis, a stable time series is necessary to avoid spurious causation. Spurious causality occurs when two non-stationary time series variables have a high correlation but no causal relationship. We use a parametric method to conduct the Phillips- Perron (PP) unit root tests (Phillips & Perron, 1988) and the augmented Dickey and Fuller's (ADF) test, which originated with Dickey and Fuller (1979).
Table-1 Results of stationarity test
Augmented Dickey Fuller (ADF) (at Level) |
|||||
With Cons |
t-Statisti |
LGDP |
LHE |
LIMR |
LLEB |
-0.55 |
4.58 |
-1.77 |
-1.4 |
||
Prob. |
0.87 |
1 |
0.39 |
0.57 |
|
Significant |
n0 |
n0 |
n0 |
n0 |
|
With Con & Trend |
t-Statisti |
-1.79 |
2.59 |
-1.35 |
1.24 |
Prob. |
0.03 |
0.01 |
0.85 |
0.03 |
|
Significant |
** |
*** |
No |
** |
|
Augmented Dickey Fuller (at First Difference) |
|||||
With Cons |
t-Statistics |
d(LGDP) |
d(LHE) |
d(LIMR) |
d(LLEB) |
-6.29 |
-2.4 |
-2.13 |
12.25 |
||
Prob. |
0 |
0.15 |
0.24 |
1 |
|
Significant |
*** |
n0 |
n0 |
n0 |
|
With Con & Trend |
t-Statisti |
-6.26 |
-4.22 |
-3.95 |
11.06 |
Prob. |
0 |
0.01 |
0.03 |
0.01 |
|
Significant |
*** |
*** |
** |
*** |
Source-Calculated by the author using E-views
Note: (*) Significant at the 10%; (**) Significant at the 5%; (***) Significant at the 1%. And (no) Not Significant
The result of an Augmented Dickey-Fuller (ADF) test in table-1 which identifies if a time series is non-stationary (has a unit root) or stationary (does not). In both cases (with constant and with constant & trend), one of the variables is stationary at the level. The p-values are higher than (0.1), so the null hypothesis (non-stationarity) cannot be rejected. At the First Difference, most variables (e.g., d(LGDP), d(LHE), d(LLFR), d(LDR)) become stationary. LGDP Stationary at the 1% level (p = 0.00). LHE and LMMR Stationary at the 5% level (with constant & trend; p = 0.03,0.05 respectively). LDR Stationary at the 1% level (p = 0.00).
Apart from the Augmented Dickey-Fuller (ADF) test, the Phillips-Perron (PP) test also evaluates each time series variable's stationarity characteristics. Unlike ADF, the PP test does not require the introduction of a lagged difference term, which is essential to choose the level of serial correlation.
Table-2 Results of Philips Perron Test of Stationarity
Philips Perron (at Level) |
|||||
With Const |
t-Statistic |
LGDP |
LHE |
LIMR |
LLEB |
-0.58 |
6.76 |
11.5 |
-1.81 |
||
Prob. |
0.86 |
1.00 |
1.00 |
0.37 |
|
Significant |
n0 |
n0 |
n0 |
n0 |
|
With Con & Trend |
t-Statistic |
-1.83 |
1.24 |
-1.29 |
5.13 |
Prob. |
0.05 |
0.03 |
0.87 |
1.00 |
|
Significant |
** |
** |
n0 |
n0 |
|
Philips Perron (at First Difference) |
|||||
With Const |
t-Statistic |
d(LGDP |
d(LHE) |
d(LIMR) |
d(LLEB) |
-6.27 |
-1.8 |
-1.27 |
12.25 |
||
Prob. |
0.01 |
0.37 |
0.63 |
1 |
|
Significant |
*** |
n0 |
n0 |
n0 |
|
With Con & Trend |
t-Statistic |
-6.24 |
-1.03 |
-2.73 |
12.2 |
Prob. |
0.00 |
0.02 |
0.02 |
0.04 |
|
Significant |
*** |
** |
** |
** |
Source-Calculated by the author using E-views
(Note: (*) Significant at the 10%; (**) Significant at the 5%; (***) Significant at the 1%. And (no) Not Significant)
The Result of the Stationarity Test using the Phillips-Perron (PP) test to ascertain the stationarity test result are given in table-2. (LGDP, LHE and LFR) is stationary at level (both 5%,10%) At level First, the difference in GDP is significant at a 1% level (p = 0.00) in both cases. GDP becomes stationary after the first differencing. Health Expenditure Significant at 5% level (p = 0.02) with constant & trend. Health expenditure becomes stationary after the first differencing. In both cases, LMMR (p = 0.03) and LEB were Significant at a 1% level (p = 0.00). It becomes stationary after first differencing. Labor Force Participation Rate Significant at 1% level (p = 0.00). It becomes stationary after first differencing. Death Rate is Significant at 1% level (p = 0.00) in both cases. It becomes stationary after first differencing.
Numerous indicators have been devised to assess the resources of health systems, including the number of physicians, hospital beds, computed tomography scanners, and health expenditures (total health expenditures per capita, health expenditures as a percentage of GDP, and health expenditures as a percentage of all expenditure in total health expenditure) (Or, 2000; Ramesh & Mirmirani, 2007; Baltagi & Moscone, 2010). The indicator utilized in this study to evaluate the health input is the total amount of health expenditures per capita. The health system results are expressed using mortality indicators (mortality rate, infant mortality rate, potential years of life lost) and longevity indicators (life expectancy at birth, life expectancy at 65, healthy life expectancy) for the overall population. When assessing a population's health, these indicators are considered reliable stand-ins (Show et al., 2002; Cutler et al., 2006; Or, 2000; Poças & Soukiazis, 2010). According to Jen et al. (2010), a nation's population is healthier if its life expectancy is higher. The study takes life expectancy into account while evaluating the state of health.
The selected lag order is specified by an asterisk ("*") in the last three columns for each criterion. For example, In the AIC column, the lag order with the lowest AIC value is considered optimal. In the LogL column, the highest value indicates a better model fit. The LR value compares the likelihood of the current model with the previous one, where higher values suggest the current model is better. Similarly, lower FPE.
Table-3 Lag Selection Criterion
Lag |
Log L |
LR |
FPE |
AIC |
SC |
HQ |
0 |
95.63 |
NA |
5.38E-06 |
-6.45 |
-6.36 |
-6.42 |
1 |
200.4 |
187.91 |
5.15E-09 |
-13.4 |
-13.12 |
-13.32 |
2 |
237.6 |
61.45* |
5.27e-10* |
-15.69* |
-15.22* |
-15.54* |
Source: Computed by Author Using E-views* Indicates lag order selected by the criterion
The Table-3 systematically evaluates various lag orders based on different model selection criteria. At lag 2, the lowest FPE, AIC, SC, and HQ values are observed, indicating superior model performance. The LR value is also highest at this Lag, suggesting significant improvement compared to the previous Lag. Therefore, lag 2 is selected as the optimal lag length for the model across most criteria.
The bound test determines whether life expectancy at birth and health spending have a long-term cointegration connection.
Table-4 Results of Bound Test
Test Statistic |
Value |
K |
|
|||
F-statistic |
9.87 |
|
||||
Significance |
10% |
5% |
1% |
|||
Sample Size |
I (0) |
I (1) |
I (0) |
I (1) |
I (0) |
I (1) |
30 |
3.3 |
3.8 |
4.09 |
4.7 |
6 |
6.8 |
Asymptotic |
3 |
3.5 |
3.62 |
4.2 |
4.9 |
5.6 |
Source: Computed by Author using EViews
The bound test and long-term relationship results are examined in Table 5.6 above. The Bound Test evaluates a long-term cointegration connection between the variables. The calculated F-statistic (9.87) is larger than the upper bound values concerning all significance levels. Thus, the null hypothesis is disproved. There is substantial evidence that the variables have a long-term cointegration relationship.
Table-5 Result of Short-Run Cointegration
Dependent Variable: D(LLEB) |
||||
Max. dependent lags: 2 (Fixed) |
||||
Fixed-lag linear regressors: LHE |
||||
Selected model: ARDL (2,2) |
||||
Variable |
Coefficient |
Std. Error |
t-Statistic |
Prob. |
COINTEQ* |
-0.37 |
0.09 |
-4.11 |
0.00 |
D (LLEB (-1)) |
2.20 |
0.70 |
21.38 |
0.00 |
D(LHE) |
0.22 |
0.08 |
2.57 |
0.00 |
D (LHE (-1)) |
0.011 |
0.009 |
1.22 |
0.00 |
R-squared |
0.95 |
Mean dependent var |
0.00 |
|
Adj R-squared |
0.95 |
SD dependent var |
0.00 |
|
S.E. of regression |
0.075 |
Akaike info criterion |
-11.08 |
|
SSR |
1.97E-05 |
Schwarz criterion |
-10.89 |
|
Log likelihood |
164.77 |
Hannan-Quinn criteria. |
-11.02 |
|
F-statistic |
198.04 |
Durbin-Watson stat |
2.1 |
|
Prob(F-statistic) |
0.00 |
|
|
Source-Authors own Calculation
Short-Run ARDL Equation: D(LLEB) = -0.37 COINTEQ + 2.20 D (LLEB (-1)) - 0.22 D(LHE) + 0.011 D (LHE (-1))
The Error Correction Technique represents the speed at which a shock is adjusted to restore equilibrium. A coefficient of (-0.37) indicates a gradual adjustment process, with each period reflecting a correction of 37% of the long-run equilibrium deviation. The lagged change in life expectancy is favourable (Coefficient (2.20), t-Statistic (21.38)) and has a highly significant impact, indicating persistence in the changes over time. First Difference of Health Expenditure (Coefficient (0.22), t-Statistic (2.57) indicates Highly significant (p = 0.00)). Changes in health spending and life expectancy have a short-term negative correlation. The short-run expansion in life expectancy is reduced by (0.22) units for every unit increase in health expenditure, presumably due to delayed effects. The Coefficient (0.01) t-statistic (3.57) is significant; (p = 0.00), according to the first Difference of Health Expenditure. In the short run, health spending and life expectancy have a statistically significant relationship. 95% of the variation can be described by the model, according to the R-squared (0.95), which indicates a very excellent fit. Adjusted R- squared (0.95). The overall model is statistically significant. Durbin-Watson Statistic (2.10),
Overall the Previous changes in life expectancy strongly influence its current changes. Immediate changes in health expenditure negatively affect life expectancy in the short run, but lagged effects are insignificant. This may indicate that the benefits of health investments require more time to manifest in improved life expectancy.
Variable * |
Coefficient |
Std. Error |
t-Statistic |
Prob. |
LHE (-1) |
0.11 |
0.013 |
8.65 |
0.00 |
C |
1.1 |
0.08 |
13.47 |
0.00 |
Note: * Coefficients derived from the CEC regression. |
Source-Authors own Calculation
ARDL Equation can be written as: Yt = 1.10 + 0.11 LHEt-1 + €t
Table-6 represents ARDL (Autoregressive Distributed Lag) model results for long-term estimation. In the long term, the dependent variable is positively and statistically significantly impacted (p < 0.05) by the lagged value of the dependent variable (LHE (-1). In the long term, the dependent variable rises by (0.11) units for every unit increase in LHE (-1). Because the constant term is statistically significant, the dependent variable will have a baseline value of (1.10) when all other variables are zero. Both variables are statistically significant at the 1% level, as indicated by their (p-values of 0.00).
According to this model, the dependent and lagged dependent variables exhibit a positive long-term relationship. The relationship is statistically robust, as shown by the low p-values and high t-statistics.
In the above figure-indicates the probability value is more than 5%, indicating that the null hypothesis of normality is not rejected. Hence, the model is usually distributed.
Null hypothesis: No serial correlation at up to 2 lags |
|||
F-statistic |
0.11 |
Prob. F (2,21) |
0.9 |
Obs*R-squared |
0.28 |
Prob. Chi-Square (2) |
0.86 |
Source- Computed by Author
The F-statistic has a probability of 0.90 (p-value), and it is 0.11. p-value is 0.86, and Obs*R-squared (0.28). The null hypothesis cannot be rejected because of the high p-values (0.90 and 0.86). This indicates that, up to two lags, the model's residuals show no indication of serial correlation. Serial correlation is absent, indicating that the model's lag structure specification is accurate.
Heteroskedasticity Test: ARCH |
|||
F-statistic |
0.36 |
Prob. F (1,26) |
0.54 |
Obs*R-squared |
0.39 |
Prob. Chi-Square (1) |
0.53 |
Source-Authors own Calculation
The Heteroskedasticity Test (ARCH Test) determines whether the variance of the residuals is homoscedastic (constant) or heteroscedastic (varies). We cannot rule out the null hypothesis since the p-value (0.54) is substantially more significant than the F- statistic (0.36). This implies that there is no discernible heteroscedasticity in the residuals. The conclusion of homoscedasticity is confirmed by a p-value of 0.53 and an Obs*R- squared of 0.39. The F-statistic and chi-square test results indicate that the model is not heteroscedastic.
There is no significant correlation between public health spending and lowering the infant and child mortality rate, according to the relationship between public health spending and IMR (Filmer & Pritchett, 1994; Musgrove, 1996). However, some believe the results may differ if the correct sample is used (Bhalotra, 2007). These authors who advocate an increase in public health spending feel that, in many instances, child and infant mortality can be brought down by spending on simple things like prenatal care, proper childbirth attendance, immunization and effective management of malnutrition and other diseases (Kumar et al., 2013) (Bhalotra, 2007). Thus, it is important to determine which school of thought is closer to the answer and whether a government initiative to increase public health spending benefits the poorer sections of society in dire need of medical facilities.
Lag |
Log L |
LR |
FPE |
0 |
75.97 |
NA |
2.09E-05 |
1 |
224.57 |
266.46 |
9.75E-10 |
2 |
237.77 |
21.83* |
5.20E-10* |
Source-Authors own Calculation
The most suitable lag length for the ARDL (Auto-Regressive Distributed Lag) model based on several factors. FPE (final prediction error), SC (Schwarz Criterion), LR (likelihood ratio), AIC (Akaike Information Criterion), HQ (Hannan-Quinn Criterion), and log L (log-likelihood). The symbol "*" signifies statistical significance for LR. Lag 2 is, therefore, chosen as the ideal lag length. Lag 2 is the ideal lag length since it exhibits a significant LR value and reduces AIC, SC, and HQ.
Table-10 Result of Bound Test and Long run relationship
Test Statistic |
Value |
K |
|
|||
F-statistic |
7.09 |
|
||||
Significance |
10% |
5% |
1% |
|||
Sample Size |
I(0) |
I(1) |
I(0) |
I(1) |
I(0) |
I(1) |
30 |
3.30 |
3.79 |
4.09 |
4.66 |
6.02 |
6.76 |
Asymptotic |
3.02 |
3.51 |
3.62 |
4.16 |
4.94 |
5.58 |
Source-Authors own Calculation
The results of the Bound Test and the Long-Term Relationship between IMR and Health Expenditure indicates that the calculated F-statistic (7.09) is greater than the upper bound values at all significance levels. The null hypothesis is no longer valid. The findings strongly suggest that the variables in the model have a long-term cointegration connection. The shared long-term trend demonstrates a stable long-term equilibrium relationship between the variables.
Variable * |
Coefficient |
Std. Error |
t-Statistic |
LHE(-1) |
-0.38 |
0.16 |
-2.3 |
C |
4.62 |
0.95 |
4.86 |
Source-Authors own Calculation
ARDL Equation can be written as:
The long-term estimation outcomes from an Autoregressive Distributed Lag (ARDL) model. Indicates that a one-unit increase in lagged LHE leads to a (0.38)-unit long-term drop in LIMR, according to the Coefficient of LHE (-1) (-0.38). The p-value (0.02) and t-statistic (-2.30) show that this Coefficient is statistically significant at the 5% level. This is the long-term relationship's intercept. According to the relationship, lagged LHE significantly lowers LIMR over time. This model suggests a negative correlation between the dependent and lagged dependent variables. The statistical robustness of the association is shown by the low p- values and high t-statistics, as well as negative causality among the lagged dependent variable and the dependent variable in the long run. The high t-statistics and low p-values indicate that the relationship is statistically robust.
Dependent Variable: D(LIMR) |
||||
x. dependent lags: 2 (Fixed) |
||||
ed-lag linear regressors: LHE |
||||
Selected model: ARDL (2,2) |
||||
Able |
efficient |
Error |
Statistic |
b. |
INTEQ* |
2 |
8 |
5 |
|
IMR (-1)) |
|
|
|
|
HE) |
5 |
8 |
7 |
|
HE(-1)) |
20 |
06 |
3 |
|
Quared |
|
an dependent var |
1 |
|
usted R-squared |
|
. dependent var |
|
|
. of regression |
31 |
ike info criterion |
78 |
|
squared resid |
E-05 |
warz criterion |
59 |
|
Likelihood |
.35 |
nan-Quinn criter. |
72 |
|
Atistic |
.32 |
bin-Watson stat |
2 |
|
b(F-statistic) |
|
|
|
Source-Authors own Calculation
The coefficient (-0.42) is negative as expected in an error-correction model. This implies that about 2% of the disequilibrium from the previous period's shock is corrected each period. The p-value (0.03) indicates marginal significance, is significant at 5%. Δ (LIMR (-1)) (0.70). indicates that 1-unit increase in the lagged first difference of LIMR leads to a (0.70) unit increase in LIMR in the short run. Highly significant (p-value = 0.00). The change in LHE and its lagged difference seem to have no significant effect on LIMR in the short run). R- squared (0.94) indicates that 94% of the variation in the dependent variable (ΔLIMR) is explained by the model. Adjusted R-squared (0.94). Similarly, high, showing the model is well-fitted. Durbin-Watson statistics (2.572) Indicates no autocorrelation issues. F-statistic (149.32). The overall model is highly significant (p-value = 0.00). The short-run dynamics indicate that the lagged dependent variable has a significant effect, but LHE and its lagged values have no immediate impact.
Source-Computed by author
Here the probability value is more than 5% which indicate that the null hypothesis of normality is not rejected. Hence, the model is normally distributed.
Breusch-Godfrey Serial Correlation LM Test: |
|||
Null hypothesis: No serial correlation at up to 2 lags |
|||
F-statistic |
10.65 |
Prob. F (2,21) |
0.91 |
Obs*R-squared |
14.6 |
Prob. Chi-Square (2) |
0.77 |
Source-Authors own Calculation
The results of the Breusch-Godfrey LM Test, often known as the Serial Correlation Test. The null hypothesis cannot be rejected since the p-value (0.91) is significantly higher than the f-statistic (10.65). This suggests no apparent serial connection in the residuals up to two lags. So, the test statistic has no serial correlation, as indicated by the Probability (0.77) and Obs*R-squared (14.60) values. The F-statistic and p-value indicate no serial correlation in the residuals.
Heteroskedasticity Test: ARCH |
|||
F-statistic |
0 |
Prob. F (1,26) |
0.92 |
Obs*R-squared |
0.01 |
Prob. Chi-Square (1) |
0.91 |
Source- Computed by Author
The Heteroskedasticity Test (ARCH Test) determines if the residuals' variance is heteroscedastic (varies) or homoscedastic (constant). We are impotent to reject the null hypothesis because the p-value (0.92) and the F-statistic = (0.00) is significantly greater than (0.05). This suggests that the residuals do not exhibit any discernible heteroscedasticity. Homoscedasticity is supported by the chi-square test p-value of 0.91 and Obs*R-squared = 0.01. The model does not exhibit heteroscedasticity, according to the findings of the chi-square test and the F-statistic. Throughout time, the residuals' variance remains constant.
CONCLUSION
The Health spending has a positive impact on India's GDP. Additionally, it comes to light that the correlation between health spending and health outcomes like-LEB, IMR, is significant and Positive for life expectancy at birth and negative for infant mortality rate. The short-run impact on life expectancy with health expenditure is positive and significant, while there is a negative impact of IMR with health expenditure in long-run. This study also concluded that economic growth (GDP) causes health expenditure and vice-versa. more awareness of the health of the people is necessary if sustainable growth is pursued; Since health disparities are essential in explaining differences in economic growth rates between states, the government must integrate health investment as a macroeconomic policy instrument. This suggests that health investment boosts economic growth.